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Somewhere around 80% of TTP is brought on by deficient action of ADAMTS13. ADAMTS13 deficiency can be constitutive, due to biallelic ADAMTS13 pathogenic variants; or acquired, due to an inhibitory antibody. Analysis of ADAMTS13 action is executed working with tests dependant on the potential of the protease to cleave normal VWF multimers in vitro (

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